Klara, my daughter will be 2 in May.  We haven’t vaccinated her at all.  I’m glad I haven’t.  She’s happy and healthy.  I’m right there with you, but I don’t want her to have any vaccinations.

Genetic testing is all fraud. It was never OUR BADDD GENES.  We all already have everything you need to get some kind of neurological syndrome, psych, or cancer infections they gave us for decades causes. Genetic testing was just a ruse to keep stealing from the public funding that should have been going to treating the cause they gave us all. 
Super-resolution imaging of virus-infected cells

revealed rod-shaped MAVS clusters on mitochondria.

These results elucidate the structural mechanism of MAVS polymerization,

and explain how an α-helical domain uses distinct chemical interactions to form self-perpetuating filaments.

DOI: http://dx.doi.org/10.7554/eLife.01489.001.
——
What those rods look like when they grow up.  
http://www.facebook.com/l.php?u=http%3A%2F%2Fmolecularalzheimer.org%2Ffiles%2FCell_wall_deficient_Bb_and_Cystic_Bb.pdf&h=pAQFvU_PX
—-
 The case for involvement of spiroplasma in the pathogenesis of transmissible spongiform encephalopathies.

http://www.ncbi.nlm.nih.gov/pubmed/24423635
J Neuropathol Exp Neurol. 2014 Feb;73(2):104-14. doi: 10.1097/NEN.0000000000000033.

Bastian FO.
Author information
Abstract
Spiroplasma biofilm formation explains the role of these wall-less bacteria in the
pathogenesis of transmissible spongiform encephalopathies (TSEs).
Spiroplasma embedded in the biofilm polysaccharide matrix are markedly
resistant to physical and chemical treatment, simulating the biologic
properties of the TSE agent. Microcolonies of spiroplasma embedded in
biofilm bound to clay are the likely mechanism of lateral transmission
of scrapie in sheep and chronic wasting disease in deer via soil
ingestion. Spiroplasma in biofilm bound to the stainless steel of
surgical instruments may also cause iatrogenic transmission of
Creutzfeldt-Jakob disease. Sessile spiroplasma in biofilm attach to the
surface by curli-like fibrils, a functional amyloid that is important
for spiroplasma entering cells.

Curli fibers have been shown to
interact with host proteins and initiate formation of a potentially
toxic amyloid that multiplies by self-assembly.

In TSE, this
mechanism may explain how spiroplasma trigger the formation of prion
amyloid. This possibility is supported by experiments that show
spiroplasma produce α-synuclein in mammalian tissue cultures. The data
linking spiroplasma to neurodegenerative diseases provide a rationale
for developing diagnostic tests for TSE based on the presence of
spiroplasma-specific proteins or nucleic acid. Research efforts should
focus on this bacterium for development of therapeutic regimens for
Creutzfeldt-Jakob disease.
—
We already have their curly fibrils growing in our skin, guts, and brains and can be found in CSP and Blood with specific DNA florescent probes.  The days of taking kids brains for seronegative infections and committing mass fraud and murder by the NIH/CDC/IDSA is over in America.